Drug class

Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics

Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics is a compendial and clinical drug classification comprising neuroprotective medications specifically indicated to slow disease progression in amyotrophic lateral sclerosis. Read more about Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics

9 drugs in this class 8 manufacturers

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Drugs in Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics

Showing 1–9 of 9 drugs

RILUZOLE

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
APOTEX CORP

RILUZOLE

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
SUN PHARMACEUTI

RILUZOLE

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
ASCEND LABORATO

TIGLUTIK (riluzole)

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
ITF PHARMA-EDW

EXSERVAN (riluzole)

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
MITSUBISHI TANA

TEGLUTIK (riluzole)

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
EDW PHARMA

RILUZOLE

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
AVKARE

RILUZOLE

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
QUINN PHARMACEU

TEGLUTIK (riluzole)

AMYOTROPHIC LATERAL SCLEROSIS AGENTS - ANTI-GLUTAMATERGICS · AMYOTROPHIC LATERAL SCLEROSIS
ITF PHARMA-EDW
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About Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics.

Amyotrophic Lateral Sclerosis Agents - Anti-glutamatergics is a compendial and clinical drug classification comprising neuroprotective medications specifically indicated to slow disease progression in amyotrophic lateral sclerosis.

The principal agent in this class, riluzole, exerts anti-glutamatergic activity by inhibiting presynaptic glutamate release, inactivating voltage-dependent sodium channels, and modulating post-synaptic glutamate receptor signaling to attenuate glutamate-mediated excitotoxicity and motor neuron injury.

Therapeutically, these agents are utilized to extend tracheostomy-free survival and delay ventilatory dependence in individuals diagnosed with amyotrophic lateral sclerosis.