Indication
Acute pulmonary thromboembolism
Acute pulmonary thromboembolism is a life-threatening cardiovascular and respiratory emergency caused by the sudden obstruction of the pulmonary arterial bed by a thrombus, which most frequently detaches and migrates from a deep vein thrombosis in the lower extremities. Read more about Acute pulmonary thromboembolism
8 drugs 7 manufacturers
Drugs that treat it
Common Acute pulmonary thromboembolism therapies.
FONDAPARINUX SODIUM
ARIXTRA (fondaparinux sodium)
FONDAPARINUX SODIUM
FONDAPARINUX SODIUM
FONDAPARINUX SODIUM
FONDAPARINUX SODIUM
FONDAPARINUX SODIUM
News & literature
Coming soonRelated codes
Coming soonRelated conditions & guidelines
Coming soonAcute pulmonary thromboembolism is a life-threatening cardiovascular and respiratory emergency caused by the sudden obstruction of the pulmonary arterial bed by a thrombus, which most frequently detaches and migrates from a deep vein thrombosis in the lower extremities.
This acute mechanical and vasoactive vascular occlusion impairs pulmonary gas exchange and increases pulmonary vascular resistance, which can precipitate right ventricular strain, dysfunction, or outright failure.
Clinically, affected individuals typically present with acute-onset dyspnea, pleuritic chest pain, tachypnea, tachycardia, or hypoxemia, with severe cases progressing to syncope, hemodynamic instability, obstructive shock, or sudden cardiac arrest.
- nih.gov
Acute pulmonary thromboembolism is a life-threatening cardiovascular and respiratory emergency caused by the sudden obstruction of the pulmonary arterial bed by a thrombus, which most frequently detaches and migrates from a deep vein thrombosis in the lower extremities
- nih.gov
Acute pulmonary thromboembolism is a life-threatening cardiovascular and respiratory emergency caused by the sudden obstruction of the pulmonary arterial bed by a thrombus, which most frequently detaches and migrates from a deep vein thrombosis in the lower extremities Clinically, affected individuals typically present with acute-onset dyspnea, pleuritic chest pain, tachypnea, tachycardia, or hypoxemia, with severe cases progressing to syncope, hemodynamic instability, obstructive shock, or sudd...
- nih.gov
This acute mechanical and vasoactive vascular occlusion impairs pulmonary gas exchange and increases pulmonary vascular resistance, which can precipitate right ventricular strain, dysfunction, or outright failure
- nih.gov
This acute mechanical and vasoactive vascular occlusion impairs pulmonary gas exchange and increases pulmonary vascular resistance, which can precipitate right ventricular strain, dysfunction, or outright failure Clinically, affected individuals typically present with acute-onset dyspnea, pleuritic chest pain, tachypnea, tachycardia, or hypoxemia, with severe cases progressing to syncope, hemodynamic instability, obstructive shock, or sudden cardiac arrest
- nih.gov
This acute mechanical and vasoactive vascular occlusion impairs pulmonary gas exchange and increases pulmonary vascular resistance, which can precipitate right ventricular strain, dysfunction, or outright failure Clinically, affected individuals typically present with acute-onset dyspnea, pleuritic chest pain, tachypnea, tachycardia, or hypoxemia, with severe cases progressing to syncope, hemodynamic instability, obstructive shock, or sudden cardiac arrest
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