Enterococcus joint infection

An Enterococcus joint infection is an uncommon form of septic arthritis or periprosthetic joint infection caused by Gram-positive catalase-negative cocci of the genus *Enterococcus*, most frequently *Enterococcus faecalis* and *Enterococcus faecium* It affects the musculoskeletal system, occurring predominantly in prosthetic joints following arthroplasty or in native joints compromised by preexisting arthropathy, invasive instrumentation, immunocompromise, or hematogenous seeding from abdominal,...

An Enterococcus joint infection is an uncommon form of septic arthritis or periprosthetic joint infection caused by Gram-positive catalase-negative cocci of the genus *Enterococcus*, most frequently *Enterococcus faecalis* and *Enterococcus faecium*

It affects the musculoskeletal system, occurring predominantly in prosthetic joints following arthroplasty or in native joints compromised by preexisting arthropathy, invasive instrumentation, immunocompromise, or hematogenous seeding from abdominal, biliary, or urinary sources

It affects the musculoskeletal system, occurring predominantly in prosthetic joints following arthroplasty or in native joints compromised by preexisting arthropathy, invasive instrumentation, immunocompromise, or hematogenous seeding from abdominal, biliary, or urinary sources Clinically, affected patients present with localized joint pain, swelling, erythema, warmth, and marked limitation of joint motion, with or without systemic fevers

It affects the musculoskeletal system, occurring predominantly in prosthetic joints following arthroplasty or in native joints compromised by preexisting arthropathy, invasive instrumentation, immunocompromise, or hematogenous seeding from abdominal, biliary, or urinary sources Pathophysiologically, the organism colonizes the synovial membrane and articular tissues—often forming recalcitrant biofilms on prosthetic hardware—which incites intense local inflammation that can lead to rapid cartilage...

Pathophysiologically, the organism colonizes the synovial membrane and articular tissues—often forming recalcitrant biofilms on prosthetic hardware—which incites intense local inflammation that can lead to rapid cartilage destruction, sinus tract formation, or implant loosening

Pathophysiologically, the organism colonizes the synovial membrane and articular tissues—often forming recalcitrant biofilms on prosthetic hardware—which incites intense local inflammation that can lead to rapid cartilage destruction, sinus tract formation, or implant loosening

Clinically, affected patients present with localized joint pain, swelling, erythema, warmth, and marked limitation of joint motion, with or without systemic fevers