Indication
Female facial hirsutism
Female facial hirsutism is an endocrine and dermatologic manifestation characterized by excessive, male-pattern growth of coarse, pigmented terminal hair on androgen-dependent areas of a female patient's face, most notably the upper lip, chin, and jawline. Read more about Female facial hirsutism
2 drugs 2 manufacturers
Drugs that treat it
Common Female facial hirsutism therapies.
URANOVIA (eflornithine HCl)
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Coming soonFemale facial hirsutism is an endocrine and dermatologic manifestation characterized by excessive, male-pattern growth of coarse, pigmented terminal hair on androgen-dependent areas of a female patient's face, most notably the upper lip, chin, and jawline.
The condition arises from androgen excess—predominantly associated with underlying disorders such as polycystic ovary syndrome or nonclassic adrenal hyperplasia—or from increased follicular sensitivity to circulating androgens mediated by 5-alpha reductase activity.
Pathophysiologically, localized androgen exposure alters the hair cycle and stimulates the transformation of fine, non-pigmented vellus hairs into thicker, darker terminal hairs.
It affects postpubertal and adult females, presenting either as an isolated idiopathic complaint or as a cardinal clinical sign of systemic hyperandrogenism.
- nih.gov
Female facial hirsutism is an endocrine and dermatologic manifestation characterized by excessive, male-pattern growth of coarse, pigmented terminal hair on androgen-dependent areas of a female patient's face, most notably the upper lip, chin, and jawline It affects postpubertal and adult females, presenting either as an isolated idiopathic complaint or as a cardinal clinical sign of systemic hyperandrogenism
- nih.gov
Female facial hirsutism is an endocrine and dermatologic manifestation characterized by excessive, male-pattern growth of coarse, pigmented terminal hair on androgen-dependent areas of a female patient's face, most notably the upper lip, chin, and jawline Pathophysiologically, localized androgen exposure alters the hair cycle and stimulates the transformation of fine, non-pigmented vellus hairs into thicker, darker terminal hairs
- nih.gov
The condition arises from androgen excess—predominantly associated with underlying disorders such as polycystic ovary syndrome or nonclassic adrenal hyperplasia—or from increased follicular sensitivity to circulating androgens mediated by 5-alpha reductase activity
- nih.gov
The condition arises from androgen excess—predominantly associated with underlying disorders such as polycystic ovary syndrome or nonclassic adrenal hyperplasia—or from increased follicular sensitivity to circulating androgens mediated by 5-alpha reductase activity It affects postpubertal and adult females, presenting either as an isolated idiopathic complaint or as a cardinal clinical sign of systemic hyperandrogenism
- endocrine.org
Pathophysiologically, localized androgen exposure alters the hair cycle and stimulates the transformation of fine, non-pigmented vellus hairs into thicker, darker terminal hairs
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