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Subependymal giant cell astrocytoma associated with tuberous sclerosis

Subependymal giant cell astrocytoma (SEGA) associated with tuberous sclerosis is a rare, slow-growing, low-grade central nervous system neoplasm that arises almost exclusively in individuals with tuberous sclerosis complex, most often presenting during childhood or adolescence. Read more about Subependymal giant cell astrocytoma associated with tuberous sclerosis

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Common Subependymal giant cell astrocytoma associated with tuberous sclerosis therapies.

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AFINITOR (everolimus)

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

Novartis Pharmaceuticals

TORPENZ (everolimus)

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

Upsher-Smith Laboratories

EVEROLIMUS

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

NORTHSTAR RX LL

EVEROLIMUS

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

BIOCON PHARMA I

EVEROLIMUS

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·SUBEPENDYMAL GIANT CELL ASTROCYTOMA ASSOCIATED WITH TUBEROUS SCLEROSIS·+1

Amneal Pharmaceuticals

AFINITOR (everolimus)

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

Novartis Pharmaceuticals

AFINITOR DISPERZ (everolimus)

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·SUBEPENDYMAL GIANT CELL ASTROCYTOMA ASSOCIATED WITH TUBEROUS SCLEROSIS·+1

Novartis Pharmaceuticals

EVEROLIMUS

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

NOVUGEN PHARMA

YULITHIRA (everolimus)

ANTINEOPLASTIC - MTOR KINASE INHIBITORS·METASTATIC RENAL CELL CARCINOMA·+6

CIVICASCRIPT, L

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About Subependymal giant cell astrocytoma associated with tuberous sclerosis.

Subependymal giant cell astrocytoma (SEGA) associated with tuberous sclerosis is a rare, slow-growing, low-grade central nervous system neoplasm that arises almost exclusively in individuals with tuberous sclerosis complex, most often presenting during childhood or adolescence.

Pathophysiologically driven by germline mutations in *TSC1* or *TSC2* that cause constitutive hyperactivation of the mechanistic target of rapamycin (mTOR) signaling cascade, the tumor typically originates in the ventricular ependymal lining near the foramen of Monro.

Although histologically benign, progressive enlargement can obstruct cerebrospinal fluid pathways, leading to obstructive hydrocephalus, elevated intracranial pressure, and associated neurological complications such as headaches, worsening seizures, and visual disturbances.

nih.gov
Subependymal giant cell astrocytoma (SEGA) associated with tuberous sclerosis is a rare, slow-growing, low-grade central nervous system neoplasm that arises almost exclusively in individuals with tuberous sclerosis complex, most often presenting during childhood or adolescence
ucsf.edu
Subependymal giant cell astrocytoma (SEGA) associated with tuberous sclerosis is a rare, slow-growing, low-grade central nervous system neoplasm that arises almost exclusively in individuals with tuberous sclerosis complex, most often presenting during childhood or adolescence Although histologically benign, progressive enlargement can obstruct cerebrospinal fluid pathways, leading to obstructive hydrocephalus, elevated intracranial pressure, and associated neurological complications such as hea...
elsevierpure.com
Subependymal giant cell astrocytoma (SEGA) associated with tuberous sclerosis is a rare, slow-growing, low-grade central nervous system neoplasm that arises almost exclusively in individuals with tuberous sclerosis complex, most often presenting during childhood or adolescence
nih.gov
Pathophysiologically driven by germline mutations in *TSC1* or *TSC2* that cause constitutive hyperactivation of the mechanistic target of rapamycin (mTOR) signaling cascade, the tumor typically originates in the ventricular ependymal lining near the foramen of Monro
nih.gov
Pathophysiologically driven by germline mutations in *TSC1* or *TSC2* that cause constitutive hyperactivation of the mechanistic target of rapamycin (mTOR) signaling cascade, the tumor typically originates in the ventricular ependymal lining near the foramen of Monro Although histologically benign, progressive enlargement can obstruct cerebrospinal fluid pathways, leading to obstructive hydrocephalus, elevated intracranial pressure, and associated neurological complications such as headaches, wo...
nih.gov
Pathophysiologically driven by germline mutations in *TSC1* or *TSC2* that cause constitutive hyperactivation of the mechanistic target of rapamycin (mTOR) signaling cascade, the tumor typically originates in the ventricular ependymal lining near the foramen of Monro
wikipedia.org
Although histologically benign, progressive enlargement can obstruct cerebrospinal fluid pathways, leading to obstructive hydrocephalus, elevated intracranial pressure, and associated neurological complications such as headaches, worsening seizures, and visual disturbances

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