Drug class

Antineoplastic - Protein-Tyrosine Kinase Inhibitors

Antineoplastic protein-tyrosine kinase inhibitors are a targeted therapeutic class of predominantly small-molecule agents designed to treat diverse malignancies. Read more about Antineoplastic - Protein-Tyrosine Kinase Inhibitors

170 drugs in this class 83 manufacturers

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Drugs in Antineoplastic - Protein-Tyrosine Kinase Inhibitors

Showing 154–162 of 170 drugs

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
SANDOZ

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
GSMS, INC.

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
EXELAN PHARMACE

IMATINIB MESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · PHILADELPHIA CHROMOSOME POSITIVE ACUTE LYMPHOBLASTIC LEUKEMIA ·+11
SAFECOR HEALTH

BOSUTINIB

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · LYMPHOID BLAST PHASE PHILADELPHIA CHROMOSOME POSITIVE CHRONIC MYELOCYTIC LEUKEMIA ·+3
ALEMBIC PHARMAC

DASATINIB

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · PHILADELPHIA CHROMOSOME POSITIVE ACUTE LYMPHOBLASTIC LEUKEMIA ·+4
NORTHSTAR RX LL

BOSUTINIB

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · LYMPHOID BLAST PHASE PHILADELPHIA CHROMOSOME POSITIVE CHRONIC MYELOCYTIC LEUKEMIA ·+3
DR.REDDY'S LAB

CAVHANZA (nilotinib)

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · CHRONIC PHASE PHILADELPHIA CHROMOSOME POSITIVE CHRONIC MYELOCYTIC LEUKEMIA ·+1
CYCLE PHARMACEU

IMATINIB MESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · PHILADELPHIA CHROMOSOME POSITIVE ACUTE LYMPHOBLASTIC LEUKEMIA ·+11
CIVICASCRIPT, L
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About Antineoplastic - Protein-Tyrosine Kinase Inhibitors.

Antineoplastic protein-tyrosine kinase inhibitors are a targeted therapeutic class of predominantly small-molecule agents designed to treat diverse malignancies.

They function by competitively inhibiting the adenosine triphosphate (ATP)–binding sites or allosteric regulatory domains of receptor and non-receptor tyrosine kinases, thereby disrupting downstream intracellular signaling cascades essential for oncogenic cell proliferation, survival, and angiogenesis.

Clinically, these agents play a foundational role in managing both hematologic neoplasms, such as chronic myeloid leukemia, and a broad range of solid tumors, including non-small cell lung cancer, renal cell carcinoma, gastrointestinal stromal tumors, and breast cancer.