CHOLESTYRAMINE LIGHT (cholestyramine/aspartame)
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA ·+1
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Drug class
Bile Sequestrants
Bile sequestrants, more formally termed bile acid sequestrants or bile acid resins, are a therapeutic class of nonabsorbable, positively charged polymeric agents. Read more about Bile Sequestrants
74 drugs in this class 34 manufacturers
Drugs in Bile Sequestrants
CHOLESTYRAMINE LIGHT (cholestyramine/aspartame)
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA ·+1
BLU PHARMACEUTI
CHOLESTYRAMINE LIGHT (cholestyramine/aspartame)
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA ·+1
PRECISION DOSE
CHOLESTYRAMINE (cholestyramine (with sugar))
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA ·+1
AJANTA PHARMA L
COLESEVELAM HCL
BILE SEQUESTRANTS · HYPERLIPIDEMIA ·+2
TRIS PHARMA INC
COLESTIPOL HCL
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA
EDENBRIDGE PHAR
CHOLESTYRAMINE (cholestyramine (with sugar))
BILE SEQUESTRANTS · HYPERCHOLESTEROLEMIA ·+1
EPIC PHARMA LLC
COLESEVELAM HCL
BILE SEQUESTRANTS · HYPERLIPIDEMIA ·+2
A-S MEDICATION
COLESEVELAM HCL
BILE SEQUESTRANTS · HYPERLIPIDEMIA ·+2
GSMS, INC.
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Bile sequestrants, more formally termed bile acid sequestrants or bile acid resins, are a therapeutic class of nonabsorbable, positively charged polymeric agents.
They exert their principal effect by binding negatively charged bile acids in the gastrointestinal tract to form insoluble complexes that are eliminated in the feces, thereby interrupting enterohepatic recirculation.
This depletion prompts the liver to convert endogenous cholesterol into new bile acids and upregulate hepatic low-density lipoprotein receptors, which lowers circulating low-density lipoprotein cholesterol.
Clinically, these agents are used primarily to treat hypercholesterolemia, improve glycemic control in type 2 diabetes mellitus, and manage chronic diarrhea caused by bile acid malabsorption.
-
nih.gov
Bile sequestrants, more formally termed bile acid sequestrants or bile acid resins, are a therapeutic class of nonabsorbable, positively charged polymeric agents They exert their principal effect by binding negatively charged bile acids in the gastrointestinal tract to form insoluble complexes that are eliminated in the feces, thereby interrupting enterohepatic recirculation
-
nih.gov
They exert their principal effect by binding negatively charged bile acids in the gastrointestinal tract to form insoluble complexes that are eliminated in the feces, thereby interrupting enterohepatic recirculation This depletion prompts the liver to convert endogenous cholesterol into new bile acids and upregulate hepatic low-density lipoprotein receptors, which lowers circulating low-density lipoprotein cholesterol Clinically, these agents are used primarily to treat hypercholesterolemia, imp...
-
nih.gov
This depletion prompts the liver to convert endogenous cholesterol into new bile acids and upregulate hepatic low-density lipoprotein receptors, which lowers circulating low-density lipoprotein cholesterol
-
nih.gov
Clinically, these agents are used primarily to treat hypercholesterolemia, improve glycemic control in type 2 diabetes mellitus, and manage chronic diarrhea caused by bile acid malabsorption
-
nih.gov
Clinically, these agents are used primarily to treat hypercholesterolemia, improve glycemic control in type 2 diabetes mellitus, and manage chronic diarrhea caused by bile acid malabsorption
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