Drug class
Hyperuricemia Agents (antimitotic)
The phrase "Hyperuricemia Agents (antimitotic)" is a compendial or database classification—principally represented by colchicine—rather than a distinct pharmacological class established by the FDA. Read more about Hyperuricemia Agents (antimitotic)
33 drugs in this class 32 manufacturers
Drugs in Hyperuricemia Agents (antimitotic)
COLCHICINE
COLCHICINE
COLCHICINE
COLCHICINE
COLCHICINE
COLCHICINE
COLCHICINE
COLCHICINE
The phrase "Hyperuricemia Agents (antimitotic)" is a compendial or database classification—principally represented by colchicine—rather than a distinct pharmacological class established by the FDA.
Agents in this grouping exert their therapeutic effects by binding to tubulin and inhibiting microtubule polymerization, which arrests cell division and suppresses neutrophil chemotaxis, degranulation, and inflammasome activation.
Although grouped under hyperuricemia therapies, these medications do not lower serum uric acid levels; instead, they serve an anti-inflammatory role primarily indicated for the treatment and prophylaxis of acute gout flares and the management of familial Mediterranean fever.
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connectrx.com
The phrase "Hyperuricemia Agents (antimitotic)" is a compendial or database classification—principally represented by colchicine—rather than a distinct pharmacological class established by the FDA
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nih.gov
The phrase "Hyperuricemia Agents (antimitotic)" is a compendial or database classification—principally represented by colchicine—rather than a distinct pharmacological class established by the FDA Although grouped under hyperuricemia therapies, these medications do not lower serum uric acid levels; instead, they serve an anti-inflammatory role primarily indicated for the treatment and prophylaxis of acute gout flares and the management of familial Mediterranean fever
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drugbank.com
Agents in this grouping exert their therapeutic effects by binding to tubulin and inhibiting microtubule polymerization, which arrests cell division and suppresses neutrophil chemotaxis, degranulation, and inflammasome activation
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litfl.com
Agents in this grouping exert their therapeutic effects by binding to tubulin and inhibiting microtubule polymerization, which arrests cell division and suppresses neutrophil chemotaxis, degranulation, and inflammasome activation Although grouped under hyperuricemia therapies, these medications do not lower serum uric acid levels; instead, they serve an anti-inflammatory role primarily indicated for the treatment and prophylaxis of acute gout flares and the management of familial Mediterranean f...
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drugs.com
Although grouped under hyperuricemia therapies, these medications do not lower serum uric acid levels; instead, they serve an anti-inflammatory role primarily indicated for the treatment and prophylaxis of acute gout flares and the management of familial Mediterranean fever
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