Drug class

Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy

The phrase "Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy" is a commercial formulary compendium category—specifically applied to pirfenidone—rather than a formal FDA-established pharmacologic class. Read more about Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy

34 drugs in this class 27 manufacturers

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Drugs in Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy

Showing 19–27 of 34 drugs

PIRFENIDONE

PULMONARY FIBROSIS TREATMENT AGENTS - ANTIFIBROTIC THERAPY · IDIOPATHIC PULMONARY FIBROSIS
SANDOZ

PIRFENIDONE

PULMONARY FIBROSIS TREATMENT AGENTS - ANTIFIBROTIC THERAPY · IDIOPATHIC PULMONARY FIBROSIS
MICRO LABS USA,

ESBRIET (pirfenidone)

PULMONARY FIBROSIS TREATMENT AGENTS - ANTIFIBROTIC THERAPY · IDIOPATHIC PULMONARY FIBROSIS
LEGACY PHARMA I

PIRFENIDONE

PULMONARY FIBROSIS TREATMENT AGENTS - ANTIFIBROTIC THERAPY · IDIOPATHIC PULMONARY FIBROSIS
AUROBINDO PHARM

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
DR.REDDY'S LAB

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
APOTEX CORP

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
CIPLA USA, INC.

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
EDENBRIDGE/DEXC

NINTEDANIB ESYLATE

ANTINEOPLASTIC - PROTEIN-TYROSINE KINASE INHIBITORS · SYSTEMIC SCLEROSIS-ASSOCIATED INTERSTITIAL LUNG DISEASE ·+2
SANDOZ
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About Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy.

The phrase "Pulmonary Fibrosis Treatment Agents - Antifibrotic Therapy" is a commercial formulary compendium category—specifically applied to pirfenidone—rather than a formal FDA-established pharmacologic class.

Clinically, these agents exert antifibrotic, anti-inflammatory, and antioxidant properties by downregulating pro-fibrotic growth factors and cytokines, notably transforming growth factor-beta (TGF-beta), thereby inhibiting fibroblast proliferation and collagen synthesis.

Their primary therapeutic role is to slow the decline of lung function and reduce disease progression in adults with idiopathic pulmonary fibrosis (IPF) and related progressive fibrosing interstitial lung diseases.