Indication

Helicobacter pylori gastritis

*Helicobacter pylori* gastritis is an infectious inflammatory disorder of the gastric mucosa caused by colonization with the microaerophilic, gram-negative bacterium *Helicobacter pylori*. Read more about Helicobacter pylori gastritis

325 drugs 142 manufacturers

Drugs that treat it

Common Helicobacter pylori gastritis therapies.

Showing 172–180 of 325 drugs

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OMEPRAZOLE MAGNESIUM

PEPTIC ULCER AGENTS·ZOLLINGER-ELLISON SYNDROME·+10

OHM LABS-SUN PH

RABEPRAZOLE (rabeprazole sodium)

PEPTIC ULCER AGENTS·MULTIPLE ENDOCRINE NEOPLASIA·+8

SARRAS HEALTH,

OMEPRAZOLE

PEPTIC ULCER AGENTS·ZOLLINGER-ELLISON SYNDROME·+10

CVS

TALICIA (omeprazole magnesium/amoxicillin trihydrate/rifabutin)

BETA-LACTAMS·DUODENAL ULCER DUE TO H. PYLORI·+2

REDHILL BIOPHAR

HELIDAC (bismuth subsalicylate/metronidazole/tetracycline HCl)

DUODENAL ULCER DUE TO H. PYLORI·+1

CASPER PHARMA L

ACIPHEX (rabeprazole sodium)

MULTIPLE ENDOCRINE NEOPLASIA·+8

WOODWARD PHARMA

NEXIUM PACKET (esomeprazole magnesium)

PEPTIC ULCER AGENTS·ZOLLINGER-ELLISON SYNDROME·+9

AstraZeneca Pharmaceuticals

PREVACID SOLUTAB (lansoprazole)

PEPTIC ULCER AGENTS·ZOLLINGER-ELLISON SYNDROME·+13

Takeda Pharmaceuticals America

PREVACID 24HR (lansoprazole)

PEPTIC ULCER AGENTS·ZOLLINGER-ELLISON SYNDROME·+13

L Perrigo Company

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About Helicobacter pylori gastritis.

*Helicobacter pylori* gastritis is an infectious inflammatory disorder of the gastric mucosa caused by colonization with the microaerophilic, gram-negative bacterium *Helicobacter pylori*.

Primarily affecting the gastrointestinal system, it is widespread across global pediatric and adult populations, often persisting indefinitely if untreated.

Pathophysiologically, bacterial virulence factors and urease production promote mucosal injury and stimulate a chronic inflammatory response marked by neutrophilic and mononuclear epithelial infiltration.

While frequently asymptomatic, the condition can present with dyspepsia or epigastric discomfort and may progress to mucosal atrophy, intestinal metaplasia, peptic ulcer disease, or gastric malignancy.

nih.gov
*Helicobacter pylori* gastritis is an infectious inflammatory disorder of the gastric mucosa caused by colonization with the microaerophilic, gram-negative bacterium *Helicobacter pylori* Primarily affecting the gastrointestinal system, it is widespread across global pediatric and adult populations, often persisting indefinitely if untreated While frequently asymptomatic, the condition can present with dyspepsia or epigastric discomfort and may progress to mucosal atrophy, intestinal metaplasia,...
nih.gov
*Helicobacter pylori* gastritis is an infectious inflammatory disorder of the gastric mucosa caused by colonization with the microaerophilic, gram-negative bacterium *Helicobacter pylori* Pathophysiologically, bacterial virulence factors and urease production promote mucosal injury and stimulate a chronic inflammatory response marked by neutrophilic and mononuclear epithelial infiltration
nih.gov
*Helicobacter pylori* gastritis is an infectious inflammatory disorder of the gastric mucosa caused by colonization with the microaerophilic, gram-negative bacterium *Helicobacter pylori*
nih.gov
Primarily affecting the gastrointestinal system, it is widespread across global pediatric and adult populations, often persisting indefinitely if untreated While frequently asymptomatic, the condition can present with dyspepsia or epigastric discomfort and may progress to mucosal atrophy, intestinal metaplasia, peptic ulcer disease, or gastric malignancy
nih.gov
Pathophysiologically, bacterial virulence factors and urease production promote mucosal injury and stimulate a chronic inflammatory response marked by neutrophilic and mononuclear epithelial infiltration
nih.gov
Pathophysiologically, bacterial virulence factors and urease production promote mucosal injury and stimulate a chronic inflammatory response marked by neutrophilic and mononuclear epithelial infiltration
nih.gov
Pathophysiologically, bacterial virulence factors and urease production promote mucosal injury and stimulate a chronic inflammatory response marked by neutrophilic and mononuclear epithelial infiltration While frequently asymptomatic, the condition can present with dyspepsia or epigastric discomfort and may progress to mucosal atrophy, intestinal metaplasia, peptic ulcer disease, or gastric malignancy

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