ZERBAXA (ceftolozane sulfate/tazobactam sodium)
NOSOCOMIAL BACTERIAL PNEUMONIA·+25
MERCK SHARP & D
Indication
Intra-abdominal Klebsiella abscess
An intra-abdominal *Klebsiella* abscess is a localized, walled-off collection of purulent exudate, necrotic tissue, and cellular debris within the abdominal cavity—most commonly involving the hepatic parenchyma, peritoneal spaces, or retroperitoneum—caused primarily by *Klebsiella pneumoniae* or related *Klebsiella* species. Read more about Intra-abdominal Klebsiella abscess
16 drugs 16 manufacturers
Drugs that treat it
Common Intra-abdominal Klebsiella abscess therapies.
CEFOXITIN (cefoxitin sodium)
STREPTOCOCCAL SEPTICEMIA·+82
APP/FRESENIUS K
CEFOXITIN (cefoxitin sodium)
BETA-LACTAMS·STREPTOCOCCAL SEPTICEMIA·+82
Hikma Pharmaceuticals USA
ZERBAXA (ceftolozane sulfate/tazobactam sodium)
BETA-LACTAMS·NOSOCOMIAL BACTERIAL PNEUMONIA·+25
Merck Sharp & Dohme
CEFOXITIN (cefoxitin sodium)
BETA-LACTAMS·STREPTOCOCCAL SEPTICEMIA·+82
APP/PREMIERPRO
CEFOXITIN (cefoxitin sodium)
BETA-LACTAMS·STREPTOCOCCAL SEPTICEMIA·+82
FRESENIUS KABI
CEFOXITIN (cefoxitin sodium)
BETA-LACTAMS·STREPTOCOCCAL SEPTICEMIA·+82
FRESENIUS KABI
Filter & compare in the drug finder →Opens the drug finder with Intra-abdominal Klebsiella abscess preselected.
News & literature
Coming soonRelated codes
Coming soonRelated conditions & guidelines
Coming soonAn intra-abdominal *Klebsiella* abscess is a localized, walled-off collection of purulent exudate, necrotic tissue, and cellular debris within the abdominal cavity—most commonly involving the hepatic parenchyma, peritoneal spaces, or retroperitoneum—caused primarily by *Klebsiella pneumoniae* or related *Klebsiella* species.
This infectious complication frequently develops secondary to gastrointestinal mucosal barrier disruption, biliary tract disease, post-surgical complications, or hematogenous translocation from the gut via the portal venous system.
Patients often present with localized abdominal pain, high fevers, leukocytosis, and systemic signs of sepsis.
Pathophysiologically, hypervirulent or multidrug-resistant capsular phenotypes can resist phagocytosis, predisposing vulnerable populations—particularly patients with diabetes mellitus, chronic hepatobiliary pathology, or recent healthcare exposures—to extensive tissue necrosis, persistent bacteremia, and potential metastatic dissemination.
- nih.gov
An intra-abdominal *Klebsiella* abscess is a localized, walled-off collection of purulent exudate, necrotic tissue, and cellular debris within the abdominal cavity—most commonly involving the hepatic parenchyma, peritoneal spaces, or retroperitoneum—caused primarily by *Klebsiella pneumoniae* or related *Klebsiella* species This infectious complication frequently develops secondary to gastrointestinal mucosal barrier disruption, biliary tract disease, post-surgical complications, or hematogenous...
- nih.gov
An intra-abdominal *Klebsiella* abscess is a localized, walled-off collection of purulent exudate, necrotic tissue, and cellular debris within the abdominal cavity—most commonly involving the hepatic parenchyma, peritoneal spaces, or retroperitoneum—caused primarily by *Klebsiella pneumoniae* or related *Klebsiella* species This infectious complication frequently develops secondary to gastrointestinal mucosal barrier disruption, biliary tract disease, post-surgical complications, or hematogenous...
- nih.gov
Patients often present with localized abdominal pain, high fevers, leukocytosis, and systemic signs of sepsis
- nih.gov
Patients often present with localized abdominal pain, high fevers, leukocytosis, and systemic signs of sepsis
- hopkinsmedicine.org
Patients often present with localized abdominal pain, high fevers, leukocytosis, and systemic signs of sepsis
- nih.gov
Pathophysiologically, hypervirulent or multidrug-resistant capsular phenotypes can resist phagocytosis, predisposing vulnerable populations—particularly patients with diabetes mellitus, chronic hepatobiliary pathology, or recent healthcare exposures—to extensive tissue necrosis, persistent bacteremia, and potential metastatic dissemination
- authorea.com
Pathophysiologically, hypervirulent or multidrug-resistant capsular phenotypes can resist phagocytosis, predisposing vulnerable populations—particularly patients with diabetes mellitus, chronic hepatobiliary pathology, or recent healthcare exposures—to extensive tissue necrosis, persistent bacteremia, and potential metastatic dissemination
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