Migraine

Migraine is a chronic neurovascular disorder of the central nervous system characterized by recurrent, disabling headache attacks that affect both pediatric and adult populations, with a pronounced female predominance

Migraine is a chronic neurovascular disorder of the central nervous system characterized by recurrent, disabling headache attacks that affect both pediatric and adult populations, with a pronounced female predominance Clinically, episodes typically manifest as moderate-to-severe, throbbing, unilateral head pain lasting 4 to 72 hours, exacerbated by physical routine and frequently accompanied by nausea, vomiting, photophobia, and phonophobia

Migraine is a chronic neurovascular disorder of the central nervous system characterized by recurrent, disabling headache attacks that affect both pediatric and adult populations, with a pronounced female predominance

Clinically, episodes typically manifest as moderate-to-severe, throbbing, unilateral head pain lasting 4 to 72 hours, exacerbated by physical routine and frequently accompanied by nausea, vomiting, photophobia, and phonophobia A subset of individuals experiences transient, reversible focal neurological disturbances known as auras, most commonly visual, which precede or accompany the cephalalgia

Clinically, episodes typically manifest as moderate-to-severe, throbbing, unilateral head pain lasting 4 to 72 hours, exacerbated by physical routine and frequently accompanied by nausea, vomiting, photophobia, and phonophobia Pathophysiologically, migraine is driven by altered brain sensory processing, cortical spreading depression, and activation of the trigeminovascular system, which triggers the perivascular release of vasoactive neuropeptides and neurogenic inflammation

A subset of individuals experiences transient, reversible focal neurological disturbances known as auras, most commonly visual, which precede or accompany the cephalalgia Pathophysiologically, migraine is driven by altered brain sensory processing, cortical spreading depression, and activation of the trigeminovascular system, which triggers the perivascular release of vasoactive neuropeptides and neurogenic inflammation

Pathophysiologically, migraine is driven by altered brain sensory processing, cortical spreading depression, and activation of the trigeminovascular system, which triggers the perivascular release of vasoactive neuropeptides and neurogenic inflammation

Pathophysiologically, migraine is driven by altered brain sensory processing, cortical spreading depression, and activation of the trigeminovascular system, which triggers the perivascular release of vasoactive neuropeptides and neurogenic inflammation