Indication
Neonatal group B streptococcal septicemia
Neonatal group B streptococcal septicemia is a life-threatening, invasive bloodstream infection occurring in newborn infants (under 28 days of age) caused by *Streptococcus agalactiae*. Read more about Neonatal group B streptococcal septicemia
88 drugs 50 manufacturers
Drugs that treat it
Common Neonatal group B streptococcal septicemia therapies.
VANCOMYCIN HCL
TYZAVAN (vancomycin HCl in water for injection (NADA,lysine))
AMPICILLIN SODIUM
VANCOMYCIN HCL
VANCOMYCIN HCL
VANCOMYCIN HCL
News & literature
Coming soonRelated codes
Coming soonRelated conditions & guidelines
Coming soonNeonatal group B streptococcal septicemia is a life-threatening, invasive bloodstream infection occurring in newborn infants (under 28 days of age) caused by *Streptococcus agalactiae*.
It primarily arises from vertical transmission during labor and delivery via aspiration of infected amniotic fluid or contact with maternal colonized vaginal secretions, though late-onset cases can also occur via postnatal community or nosocomial acquisition.
Pathophysiologically, the bacteria proliferate intravascularly and trigger systemic inflammatory dysregulation, presenting clinically with temperature instability, lethargy, poor feeding, respiratory distress, and rapid progression to cardiovascular collapse, septic shock, or secondary meningitis.
- medlineplus.gov
Neonatal group B streptococcal septicemia is a life-threatening, invasive bloodstream infection occurring in newborn infants (under 28 days of age) caused by *Streptococcus agalactiae*
- nih.gov
Neonatal group B streptococcal septicemia is a life-threatening, invasive bloodstream infection occurring in newborn infants (under 28 days of age) caused by *Streptococcus agalactiae*
- nih.gov
It primarily arises from vertical transmission during labor and delivery via aspiration of infected amniotic fluid or contact with maternal colonized vaginal secretions, though late-onset cases can also occur via postnatal community or nosocomial acquisition Pathophysiologically, the bacteria proliferate intravascularly and trigger systemic inflammatory dysregulation, presenting clinically with temperature instability, lethargy, poor feeding, respiratory distress, and rapid progression to cardio...
- aap.org
Pathophysiologically, the bacteria proliferate intravascularly and trigger systemic inflammatory dysregulation, presenting clinically with temperature instability, lethargy, poor feeding, respiratory distress, and rapid progression to cardiovascular collapse, septic shock, or secondary meningitis
- pedemmorsels.com
Pathophysiologically, the bacteria proliferate intravascularly and trigger systemic inflammatory dysregulation, presenting clinically with temperature instability, lethargy, poor feeding, respiratory distress, and rapid progression to cardiovascular collapse, septic shock, or secondary meningitis
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