AQUASOL A (vitamin A palmitate)
VITAMIN A DEFICIENCY
HOS-PFIZ/CASPER
Indication
Vitamin A deficiency
Vitamin A deficiency is a systemic nutritional disorder caused by prolonged inadequate dietary intake or impaired absorption, storage, and transport of fat-soluble retinoids. Read more about Vitamin A deficiency
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Common Vitamin A deficiency therapies.
AQUASOL A (vitamin A palmitate)
VITAMIN A·VITAMIN A DEFICIENCY
RISING PHARM
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Coming soonVitamin A deficiency is a systemic nutritional disorder caused by prolonged inadequate dietary intake or impaired absorption, storage, and transport of fat-soluble retinoids.
It most commonly affects infants, young children, and pregnant or lactating individuals in resource-limited regions, as well as individuals with chronic malabsorptive or hepatobiliary disorders.
Pathophysiologically, the loss of vitamin A impairs retinal rhodopsin regeneration, compromises mucociliary epithelial integrity, and disrupts immune function.
Clinically, it manifests as xerophthalmia—progressing from nyctalopia (night blindness) and conjunctival Bitot spots to corneal xerosis, ulceration, and potentially irreversible keratomalacia—alongside increased vulnerability to severe infectious morbidity and follicular hyperkeratosis.
- nih.gov
Vitamin A deficiency is a systemic nutritional disorder caused by prolonged inadequate dietary intake or impaired absorption, storage, and transport of fat-soluble retinoids It most commonly affects infants, young children, and pregnant or lactating individuals in resource-limited regions, as well as individuals with chronic malabsorptive or hepatobiliary disorders
- msdmanuals.com
Vitamin A deficiency is a systemic nutritional disorder caused by prolonged inadequate dietary intake or impaired absorption, storage, and transport of fat-soluble retinoids It most commonly affects infants, young children, and pregnant or lactating individuals in resource-limited regions, as well as individuals with chronic malabsorptive or hepatobiliary disorders Pathophysiologically, the loss of vitamin A impairs retinal rhodopsin regeneration, compromises mucociliary epithelial integrity, an...
- nih.gov
It most commonly affects infants, young children, and pregnant or lactating individuals in resource-limited regions, as well as individuals with chronic malabsorptive or hepatobiliary disorders
- nih.gov
Pathophysiologically, the loss of vitamin A impairs retinal rhodopsin regeneration, compromises mucociliary epithelial integrity, and disrupts immune function
- who.int
Clinically, it manifests as xerophthalmia—progressing from nyctalopia (night blindness) and conjunctival Bitot spots to corneal xerosis, ulceration, and potentially irreversible keratomalacia—alongside increased vulnerability to severe infectious morbidity and follicular hyperkeratosis
- researchgate.net
Clinically, it manifests as xerophthalmia—progressing from nyctalopia (night blindness) and conjunctival Bitot spots to corneal xerosis, ulceration, and potentially irreversible keratomalacia—alongside increased vulnerability to severe infectious morbidity and follicular hyperkeratosis
- dermnetnz.org
Clinically, it manifests as xerophthalmia—progressing from nyctalopia (night blindness) and conjunctival Bitot spots to corneal xerosis, ulceration, and potentially irreversible keratomalacia—alongside increased vulnerability to severe infectious morbidity and follicular hyperkeratosis
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